New Evidence for Causal Central Mechanism of Hyperglycemia in Subarachnoid Hemorrhage Secondary to Ischemic Degenerative Disruption of Circuitry Among Insular Cortex, Nodose Ganglion, and Pancreas: Experimental Study

AYDIN, Mehmet; Kanat, AYHAN; Aydin, Nazan; KANTARCI, Abdulmecit; Ayvaz, Muhammet; Rakici, HALİL; Yolas, Coskun; Kepoglu, Umit; DEMİRCİ, Elif

doi:10.1016/j.wneu.2017.06.176

New Evidence for Causal Central Mechanism of Hyperglycemia in Subarachnoid Hemorrhage Secondary to Ischemic Degenerative Disruption of Circuitry Among Insular Cortex, Nodose Ganglion, and Pancreas: Experimental Study

AYDIN M. D., Kanat A., Aydin N., KANTARCI A., Ayvaz M. A., Rakici H., ...Daha Fazla

WORLD NEUROSURGERY, cilt.106, ss.570-577, 2017 (SCI-Expanded, Scopus)

Yayın Türü: Makale / Tam Makale
Cilt numarası: 106
Basım Tarihi: 2017
Doi Numarası: 10.1016/j.wneu.2017.06.176
Dergi Adı: WORLD NEUROSURGERY
Derginin Tarandığı İndeksler: Science Citation Index Expanded (SCI-EXPANDED), Scopus
Sayfa Sayıları: ss.570-577
Anahtar Kelimeler: Hyperglycemia, Insular cortex, Nodose ganglion and pancreas, Subarachnoid hemorrhage, MIDDLE CEREBRAL-ARTERY, GLOSSOPHARYNGEAL NERVE, 1ST REPORT, ACUTE HYDROCEPHALUS, CAROTID-BODY, RISK-FACTORS, VAGUS NERVE, VASOSPASM, VAGAL, RAT
Recep Tayyip Erdoğan Üniversitesi Adresli: Evet

Özet

INTRODUCTION: Although hyperglycemia is a serious complication of subarachnoid hemorrhage, its pathophysiologic mechanism based on neural circuitry has not been known. -