The independent relationship between systemic inflammation and fragmented QRS complexes in patients with stable angina pectoris

Cetin M., KOCAMAN S. A., CANGA A., DURAKOĞLUGİL M. E., ERDOĞAN T., ŞATIROĞLU Ö., ...Daha Fazla

KARDIOLOGIA POLSKA, cilt.70, sa.7, ss.668-676, 2012 (SCI-Expanded) identifier identifier identifier

  • Yayın Türü: Makale / Tam Makale
  • Cilt numarası: 70 Sayı: 7
  • Basım Tarihi: 2012
  • Dergi Adı: KARDIOLOGIA POLSKA
  • Derginin Tarandığı İndeksler: Science Citation Index Expanded (SCI-EXPANDED), Scopus
  • Sayfa Sayıları: ss.668-676
  • Anahtar Kelimeler: fragmented QRS, fQRS, coronary artery disease, cardiovascular risk, inflammation, left ventricular dysfunction, coronary atherosclerotic burden, C-REACTIVE PROTEIN, CORONARY-ARTERY-DISEASE, BUNDLE-BRANCH BLOCK, MYOCARDIAL-INFARCTION, DILATED CARDIOMYOPATHY, CARDIOVASCULAR-DISEASE, CARDIAC FIBROSIS, ANATOMIC BASIS, HEART-FAILURE, QTC INTERVAL
  • Recep Tayyip Erdoğan Üniversitesi Adresli: Evet

Özet

Background: QRS complex fragmentations can frequently be seen on routine ECG with narrow or wide QRS complex. Fragmented QRS complexes (fQRS) are defined as various RSR' patterns (>= 1 R' or notching of S wave or R wave) in two contiguous leads corresponding to a major coronary artery territory. In previous studies, fQRS has been associated with increased morbidity and mortality, sudden cardiac death and recurrent cardiovascular events. The causative relationship between fQRS and cardiac fibrosis has been shown, but it has not been extensively studied whether there are different mechanisms for the development of fQRS.